Lipofuscin & Heavy Metals
By Atom Bergstrom
Re: As early as 1912, progressive accumulation of lipofuscins concurently with aging was recognized in animal tissues.
Capable scientists (giving them the benefit of the doubt) aren’t capable historians because lipofuscins were already linked with aging a hundred years earlier.
Take it from The Timely Atom, who discovers many cases of Medical Amnesia while skimming through supposedly “outdated” books.
I paid attention to my mentor, Adano Ley (Swami Nitty-Gritty) when he said (more than several times) …
“There’s nothing new under the Sun.”
Lipofuscin and heavy metals go together like death and taxes.
But which is primary, the aging pigment or the aging metal?
A.J. Rawson, G.W. Patton, S. Hofmann, G.G. Pietra, & L. Johns (“Liver abnormalities associated with chronic mercury accumulation in stranded Atlantic bottlenose dolphins,” Ecotoxicology and Environmental Safety, Feb. 1993) wrote …
“Eighteen stranded Atlantic bottlenose dolphins (Tursiops truncatus) examined postmortem were sampled for histologic study. All cases were examined for ferric ion and lipofuscin. Ages were determined from tooth growth layers. Electron microscopic (EM) examination and X-ray spectroscopy (EDAX) were performed. Chemical analysis for mercury was conducted on 12 of the animals by atomic absorption spectrophotometry. Nine animals were found to have excessive lipofuscin in both liver and kidney. Four of these nine animals also exhibited active liver disease (fat globules, central necrosis, lymphocytic infiltrates) whereas, of the animals without the excessive pigment, only one animal had an active liver lesion. EM and EDAX showed electron-dense amorphous material presumably within lysosomes to be Hg with no deposits on mitochondrial or nuclear membranes noted. Age relationship to portal pigment deposition was positive. Liver mercury concentrations ranged from 0.01 to 443 micrograms/g of wet weight with all animals having liver pigment yielding values of or above 61 micrograms/g, whereas all animals lacking pigment had values of or below 50 micrograms/g. The evidence suggests that the excessive pigment accumulation is related to toxic effects of Hg and presents as increased active liver disease.”
So, is the mercury in your dental fillings triggering lipofuscin to fast-forward aging and dementia?
Or is it the other way around?
Or is it a mutual added-aging society?