Sterols Are Yang & Fatty Acids Are Yin
My blog series (and future e-book) on Gold Trauma and its spawning of the Gold Standard (Screw the Golden Years) will continue at a later date.
Meanwhile, let’s bang a U-ie and drive back to Yellow Fat Disease and polyunsaturated fatty acids.
Emanuel Revici, M.D. (Research In Physiology As Basis Of Guided Chemotherapy: With Special Application To Cancer, 1961) wrote …
“When a sample of ordinary venous blood is treated with a preparation of cholesterol or nonsaponifiable fractions, using the above mentioned technique [see pages 666 & 667] and after its separation from the cholesterol it is agitated with air or oxygen which is passed through these samples, the color becomes a bright vermilion red, and this persists for a long time. When the same venous blood is treated with a preparation of polyunsaturated fatty acids as mentioned above, the color becomes very dark, almost black purple. When air or oxygen is passed through these samples, the blood becomes lighter in color for only a short time, the darker color reappearing within a few minutes. One is immediately impressed by the similarity of the cholesterol-treated blood to arterial blood, while the fatty acid-treated blood is similar to venous blood, and especially to the color of venous blood in cases of shock.”
According to one point of view …
Cholesterol is anti-inflammatory, required 80 percent of the time.
Polyunsaturated fatty acids (PUFAs) are pro-inflammatory, required 20 percent of the time.
Overall food content — on and in the ground, not in the sea — provides PUFAs in the following ratio …
Omega-3 compared to omega-6 fatty acids — 1 to 11 parts (not 1 to 1, as fancied by certain “experts” like Andrew Stoll and Susan Allport).
According to Dr. Revici (continued from his textbook) …
“The sterol-richer red cells appear capable of retaining for a longer period of time, the amount of oxygen which hemoglobin has fixed, while a rapid reduction in oxyhemoglobin is seen in the red blood cells when the polyunsaturated acids intervene. This led us to consider an intervention of these two groups of lipids in relation to the oxygen transportation by the red cells. Bearing in mind the fact that while cholesterol reduces cell permeability and polyunsaturated fatty acids increase it, an alternating intervention of these lipids seems to play a role in a better distribution of oxygen. The oxygen which is fixed by hemoglobin when the red blood cells have passed the lungs, is largely retained as such by the intervention of the sterols until they reach the point in the tissues where liberation of oxygen is necessary, this being favored now by the intervention of the fatty acids.”
What does this tell us about leaky gut syndrome?
Well, WHAT leaks into WHERE because of increased cell permeability?
The toxic abnormal PUFAs leak into the fluid matrix of the body — blood, lymph, bile, gastric juice, mucus, saliva, tears, and sweat.
The PUFAs prompted by all stages of clinical shock are too alkaline to be balanced by cholesterol.
So what acid is produced to balance this abnormal alkalinity?
Oxalic acid is produced — a little dab’ll do ya, but too much is toxic to mucus membranes and the upper respiratory tract.