Hemorrhages & Omega 3 Disease

By Atom Bergstrom

Atom’s Blog

Petechiae (puh-TEE-kee-ee) have many causes.

Omega 3 Disease is an underlying cause you won’t find listed by Wikipedia, JAMA, Medline, Mayo Clinic, or the rest of the usual sources.

Mainstream Monolithic Medicine restricts Truth to a need-to-know basis, so truth-finding is always a Black Bag Job.

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A Yellow Fat Disease by any other name is still a Yellow Fat Disease.

Add Hitra Disease to the other names of Omega 3 Disease.

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M. Fjølstad & A.L. Heyeraas (“Muscular and myocardial degeneration in cultured Atlantic salmon, Salmo salar L., suffering from ‘Hitra disease’,” Journal of Fish Diseases, Jul. 1985) wrote …

“In this investigation of the condition known currently as ‘Hitra disease’, anaemia was usually seen and transudates to the peritoneal cavity and pericardial sac were often found. Muscular and myocardial degeneration were constant features and deposits of ceroid were sometimes found in the livers. Compared to similar conditions in fish and in higher animals it is reasonable to suggest that the main cause of the disease may be related to vitamin E/selenium deficiency and diets high in polyunsaturated fatty acids. The levels of unsaturated fatty acids in particular would appear of great importance in the formulation of diets for cultured salmon.”

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Trygve T. Poppe, Tore HÃ¥stein, Arne Frøslie, Nils Koppang, & Gunnar Norheim (“Nutritional aspects of Hemorrhagic Syndrome (‘Hitra Disease’) in farmed Atlantic salmon Salmo salar,” Diseases of Aquatic Organisms, October 15, 1986) wrote …

“Since 1977, Haemorrhagic Syndrome (HS) or ‘Hitra Disease’ has been the most important and widespread disease in farmed Atlantic salmon Salmo salar L. in Norway (Poppe et al. 1985b).”

According to the same source …

“The most severe histopathological changes in diseased fish were found in the skeletal and heart musculature. In other organs congestion and oedema were frequently found, as well as focal haemorrhages in some cases. Moderate hepatocellular fatty infiltration and focal liver degeneration were typical findings. Congestion and subcapsular (superficial) haemorrhages were also frequently found, although the liver tissue was otherwise without visible pathological changes. No ceroid-like material was observed.

“Severe signs of circulatory failure with congestion, peritubular oedema and degeneration could be found in the kidney as well as haemorrhages in the haematopoietic tissue. Focal haemorrhages and clusters of bacteria were frequently found in the spleen. Congestion, oedema, focal hemorrhages and bacteria were frequently seen in the subcutaneous connective tissue. Similar changes together with desquamation of the epithelium were found in the digestive tract.”

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Many typical Yellow Fat Disease changes are listed in the above article, including …

“congestion, oedema, exudative and haemorrhagic diathesis, anaemia, microangiopathy, muscular and myocardial dystrophy and/or degeneration (‘mulberry heart disease’), focal liver degeneration (‘hepatosis dietetica’), steatitis (‘yellow fat’) and catarrhal enteritis.”

Last but not least, Muscular Dystrophy.