Kittens & Yellow Fat Disease

By Atom Bergstrom

Atom’s Blog

According to the National Research Council (Nutrient Requirements of Dogs and Cats, 2006) …

“Because of the nature of the diets consumed by cats, they are more susceptible than dogs to vitamin E deficiency. Cat diets, especially canned, are typically higher in fat than dog diets and are frequently based on fish products, especially tuna, containing long-chain PUFAs that are prone to lipid peroxidation. Cordy (1954) reported that 20 or 40 mg of all-rac-α-tocopherol per day protected kittens given a commercial cat food composed mainly of fish from the development of steatitis [Yellow Fat Disease], whereas 10 mg of all-rac-α-tocopherol was inadequate. Gershoff and Norkin (1962) gave kittens a basal purified diet containing 200 g.kg–1 of vitamin E-free lard without vitamin E supplementation. Steatitis [Yellow Fat Disease] was not observed in any of the kittens receiving this basal diet, even in the absence of vitamin E. However, on histological examination, four of the six kittens were found to have focal myositis of the skeletal muscles and myocarditis. Supplementation of the basal diet with 17 IUs of d-α-tocopherol.kg–1 decreased the incidence of myositis to one cat in four, whereas addition of 34 or 68 IUs of vitamin E.k–1 prevented lesions in all cats. When 50 g of tuna oil was substituted for lard in the diet, cats without vitamin E supplementation developed severe steatitis [Yellow Fat Disease]. When 34 or 68 IUs of vitamin E.k–1 were added to the tuna oil-supplemented diet, the severity of steatitis [Yellow Fat Disease] was diminished and it was totally absent when 136 IUs of vitamin E were added to the tuna oil diet.”

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What is meant by “long-chain PUFAs”?

The National Research Council is referring to eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA).

The same applies to alpha-linolenic acid (ALA), but it played no part in the studies cited above.

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What is myositis?

It’s inflammation and degeneration of muscle tissue.

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What is myocarditis?

It’s inflammation and degeneration of the heart muscle, most often attributed (WRONGLY) to a bacterial infection.

Meanwhile, the Assassins In White carry on their intensive campaign against vitamin E.

Here’s a classic example from WebMD.

Daniel J. DeNoon (Vitamin E Harms More Than It Helps Supplement No Help for Cancer, Heart, Stroke — but Increases Heart Failure Risk,” webmd dot com, Mar. 15, 2005) wrote …

“Vitamin E was linked to a 13% higher risk of heart failure and a 21% increased risk of hospitalization for heart failure. That’s a pretty small risk. But since the vitamin did no good at all, it’s a risk not worth taking.”

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What is all-rac-α-tocopherol?

It’s roll up your pants, it’s too late to save your shoes!

The manure gets very deep when it comes to legitimate supplement info.

All-rac-α-tocopherols defined as “a mixture of eight forms (isomers) of alpha-tocopherol.”

Four of these eight isomers — the tocotrienols — weren’t synthesized from rubber until 1964, so how does the National Research Council explain the two studies cited in 1954 and 1962 — ten years and two years BEFORE THEY WERE DISCOVERED.

The tocotrienols are a total scam, designed to keep you from protecting yourself from heart disease, cancer, and stroke with authentic forms of vitamin E.

Ray Peat (“Vitamin E: Estrogen antagonist, energy promoter, and anti-inflammatory,” 2006-2016) wrote …

“The unsaturated tocotrienols have hardly been tested for the spectrum of true vitamin E activity, and animal studies have suggested that it may be toxic, since it caused liver enlargement.”

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Vitamin E isn’t merely an antioxidant.

It’s an ANTI-ESTROGEN, something the “estrogen industry” has been hiding since the 1940s, when it started its SMEAR CAMPAIGN.

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What does “histological examination” mean?

It means we should honor all the innocent kittens sacrificed just so we could know a few facts about vitamin E.
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